TypeWOAHStIKo VetLit.127
DNA virus (alphaherpesvirus, genus Varicellovirus)
Fact sheets for horse owners
The 32 pathogens on a typical lab request form – what they are, how they spread, what to look out for and how to protect your horse.
Pathogen list and test methods taken from page 2 (“Testing for Equine Infectious Diseases”) of the request form “Request Form Infection” by Labor Dr. Böse GmbH, Harsum (DE), version 2025-11-09. Independent educational material – not issued by or affiliated with the laboratory. Facts checked against WOAH and FLI where they cover the topic; other sources are labelled. Status: October 2026.
These sheets do not replace veterinary advice. If your horse is ill, call your vet.
Legal status sources: WOAH Terrestrial Animal Health Code, chapter 1.3 (edition 2024) · Tierseuchenmeldeverordnung (TierSeuchMeldV) of 10 March 2026, in force since 11 March 2026. Per-pathogen sources are listed on each sheet.
Section 1
Infections that can make a pregnant mare lose her foal. Samples come from the afterbirth discharge (lochia), the uterus lining or the aborted foal.
| Aerobic culture incl. resistograms | culture swab | #5020 |
Sampling note on the form: PCR: dry swab, cooled 24 h · BE aerobic: culture swab · sample sites: lochia, endometrium, lung puncture aspirate (foetal), others
Equine herpesvirus 1 · Varicellovirus equidalpha1 (Equid alphaherpesvirus 1)
A very common horse herpesvirus. It causes airway infections, abortion storms in pregnant mares and a dangerous neurological form, and hides in the body for life.
DNA virus (alphaherpesvirus, genus Varicellovirus)
Fever after 1–2 days, other signs 2–8 days; abortion weeks to months later
No known risk. WOAH reports no evidence that EHV-1 poses any health risk to people, including laboratory staff working with the virus.
Mainly by close contact and breathing in droplets from coughing or snorting horses; also via contaminated clothing, tack and buckets. Aborted foetuses, afterbirth and birth fluids carry huge amounts of virus. Latent carriers can restart shedding.
| Method | Sample | Form code |
|---|---|---|
| PCRAbortion · Respiratory · CNS | dry swab (cooled), EDTA blood; abortion: lochia, endometrium, foetal lung aspirate | #1912 |
| PCR panels with EHV-4 (+ EVA / + influenza)Abortion · Respiratory | dry swab (cooled) | #8620 / 8660 / 8720 / 8760 |
| VNT (antibodies)CNS | serum, preferably paired samples | #1910 |
Vaccines reduce airway disease and abortion but do not prevent the neurological form. Infected horses carry the virus for life. During outbreaks, take temperatures twice daily.
Equine herpesvirus 4 · Varicellovirus equidalpha4 (Equid alphaherpesvirus 4)
The most common viral cause of airway infection in horses. Usually a mild fever and runny nose, rarely abortion. Like EHV-1, it stays in the body for life.
DNA virus (alphaherpesvirus, genus Varicellovirus)
Usually 2–8 days; fever may start within 1–2 days
No known risk. WOAH reports no evidence that EHV-4 poses any health risk to people, including laboratory staff working with the virus.
Breathing in droplets and close contact with infected horses, plus contaminated tack, buckets and clothing. Common at shows and where young horses mix. Carriers can reactivate and shed the virus under stress.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab (cooled), EDTA blood; abortion: lochia, endometrium, foetal lung aspirate | #1942 |
| PCR panels with EHV-1 (+ EVA / + influenza) | dry swab (cooled) | #8620 / 8660 / 8720 / 8760 |
Most horses are infected young and reinfected repeatedly. EHV-4 stays dormant in a nerve cluster in the head and can reactivate under stress.
Equine viral arteritis · Alphaarterivirus equid (equine arteritis virus, EAV)
A contagious virus spread by breathing and by breeding. Often silent, it can cause fever, swelling and abortion, and some stallions shed it in semen for years.
Enveloped RNA virus (arterivirus, family Arteriviridae)
Usually 3–10 days (range 2–14), shorter after airborne spread
No. WOAH states the virus does not present a human health hazard, and there is no evidence that it infects people.
By droplets from acutely infected horses and by mating or artificial insemination with infected semen. Carrier stallions are the main reservoir. Urine, aborted foetuses, afterbirth and contaminated equipment can also spread it.
| Method | Sample | Form code |
|---|---|---|
| PCRAbortion / Respiratory / Additional | dry swab (cooled), EDTA blood (cooled) | #1502 |
| PCR panel 2 (EHV-1, EHV-4, EVA)Abortion | dry swab (cooled) | #8660 |
| VNT (antibodies)Additional | serum | #1500 |
Mares, geldings and young colts do not become carriers. Vaccinated horses test antibody-positive, which can block exports; the vaccine is not advised in pregnant mares.
Leptospira spp. (pathogenic species, e.g. Leptospira interrogans, Leptospira kirschneri)
A bacterial infection spread mainly in urine. In horses it is linked to abortion and recurrent eye inflammation (moon blindness). People can catch it too.
Spiral-shaped bacterium (spirochaete) with many serovars
2–20 days to fever; abortion 2–4 weeks later; eye disease months–years later
Yes. People are infected through urine-contaminated water, soil or animal contact entering skin cuts or mucous membranes. Farm workers, vets and water-sport enthusiasts are most at risk; illness ranges from flu-like to severe.
Contact with urine of infected rodents, wildlife or horses, often via stagnant water, wet soil or flooded pasture, or with aborted tissue and birth fluids. Recovering horses can shed bacteria in urine for up to 3 months.
| Method | Sample | Form code |
|---|---|---|
| PCRAbortion / Additional | dry swab (cooled), EDTA blood, tissue, urine°, aqueous humour° | #1622 |
| MAT (antibodies)Additional | serum | #1600 |
Leptospira can survive up to 10 months in water but is killed by drying and common disinfectants. Moon blindness tends to flare repeatedly and can end in blindness.
Equine chlamydiosis · Chlamydia psittaci (main species found in equine abortion; other Chlamydia species rarely)
A bird-associated bacterium that can cause late abortion and fatal illness in newborn foals. Infected placentas can give people psittacosis, a serious pneumonia.
Tiny bacterium that multiplies only inside host cells
Unknown in horses; in people usually 5–14 days, sometimes longer
Yes. Five vet-school staff and students caught psittacosis from equine foetal membranes; further cases are suspected. Infection is by inhalation and causes fever, headache and pneumonia. Foaling staff and vets are most exposed.
Horses probably catch it from infected birds (in Australia, wild parrots), likely via droppings or dust. People are infected by inhaling material from infected placentas, aborted or sick newborn foals, or bird droppings.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab (cooled) – lochia, endometrium, foetal lung aspirate | #5062 |
C. psittaci has also been found in equine abortions in Europe, including Germany. Dried bird material can stay infectious for about four weeks. Australian cases cluster in winter and spring.
Section 2
Coughing, nasal discharge and fever – the classic yard outbreaks. Nasal or throat swabs and blood samples help to find the cause.
Equine influenza virus (equine flu) · Alphainfluenzavirus influenzae (influenza A virus, subtype H3N8)
Highly contagious 'horse flu' that spreads fast through yards and events. Rarely fatal, but causes fever, harsh cough and weeks off work; vaccination is core in Germany.
RNA virus (influenza A, H3N8; family Orthomyxoviridae)
1–3 days; can be under 24 hours
Very unlikely. Equine flu has not been shown to cause illness in people; antibodies were found mainly in people exposed through their work. It has spread to dogs.
Breathing in droplets from coughing horses (airborne up to about 40 m) and close contact. People, vehicles, tack and buckets can carry it. Vaccinated horses may shed virus without showing signs.
| Method | Sample | Form code |
|---|---|---|
| HI (antibodies), A equi II European and American type | serum | #2055, 2060 |
| PCR | dry swab (cooled) | #2075 |
| PCR panel 2 (EHV-1, EHV-4, influenza) | dry swab | #8760 |
Rest at least 3 weeks after illness, about one week per day of fever, because damaged airways need around 3 weeks to heal.
Streptococcus equi subsp. equi
A highly contagious bacterial infection of the throat and head lymph nodes. It spreads quickly through yards, and some recovered horses silently carry it for years.
Gram-positive bacterium (Lancefield group C streptococcus), horse-adapted
Usually 3–14 days until fever (StIKo Vet: mostly 3–8 days)
Very rarely. Isolated human infections have been reported, mainly in people with weakened immunity, who should avoid contact with sick horses. Strangles is essentially a disease of horses, donkeys and mules.
Direct contact and droplets from nasal discharge or coughing, and indirectly via shared water troughs, buckets, tack, grooming kit, clothing and hands. Horses that are incubating, recovering or silent carriers can all spread it.
| Method | Sample | Form code |
|---|---|---|
| Culture (BE aerobic) + PCR + resistogram | culture swab | #5099 |
| PCR | culture swab | #5093 |
| ELISA (antibodies) | serum | #5089 |
| Panel S. equi (equi + zooepidemicus): culture + PCR + resistogram | culture swab | #5094 |
About 1 in 10 infected horses become long-term carriers in the guttural pouches (air sacs behind the throat). Bacteria survive weeks in water but only days on dry surfaces.
Streptococcus zooepidemicus · Streptococcus equi subsp. zooepidemicus
A normal resident of horses' airways and genital tract that causes disease when defences drop. It is a leading cause of pneumonia, womb infection and abortion in horses.
Gram-positive bacterium (Lancefield group C streptococcus), opportunist
Not defined: opportunistic infections appear when the horse's defences drop
Rarely, but infections can be severe (meningitis, blood poisoning, joint infection). Cases mostly involve people in close daily contact with horses, sometimes with weakened immunity. Wash hands; avoid nose-to-nose contact.
Most infections come from the horse's own bacteria after stress, transport, viral infection or injury. Outbreaks between horses occur occasionally, and people in close contact can rarely be infected.
| Method | Sample | Form code |
|---|---|---|
| Panel S. equi (equi + zooepidemicus): culture + PCR + resistogram | culture swab | #5094 |
Healthy horses commonly carry it, so laboratories must distinguish it from the strangles bacterium, a closely related subspecies that is far more contagious.
Foal pneumonia · Rhodococcus equi (conserved name; synonyms Prescottella equi, Rhodococcus hoagii)
A soil bacterium causing slow-developing pneumonia with lung abscesses in foals aged 1–6 months. It is common on some breeding farms and early signs are easily missed.
Gram-positive soil bacterium that survives inside immune cells
Infected in first weeks of life; signs usually at 1–6 months
Rarely. It can cause severe lung or whole-body infection in people with weakened immunity (e.g. HIV, transplant patients), usually from soil or manure. Infection in healthy people is extremely rare.
Foals inhale dust containing virulent bacteria in their first weeks of life. The bacteria live in soil and herbivore manure; sick foals shed large numbers in droppings. It does not spread directly between foals.
| Method | Sample | Form code |
|---|---|---|
| Culture (BE aerobic) + PCR + resistogram | culture swab | #5053 |
Survives over a year in soil. Adult horses are rarely affected unless immunocompromised. Many infected foals recover without treatment; unnecessary antibiotics promote resistant strains.
Section 3
Infections of the brain and spinal cord: wobbliness, weakness, behaviour changes. Always an emergency for your vet.
Orthobornavirus bornaense (Borna disease virus 1, BoDV-1)
A rare, usually fatal brain inflammation of horses in parts of Germany, Austria, Switzerland and Liechtenstein. The virus comes from a wild shrew; infected horses do not pass it on.
Enveloped negative-sense RNA virus (orthobornavirus) that attacks the brain
About 1–3 months
People can develop fatal brain inflammation, but very rarely (about 5–10 cases a year in Germany, mostly rural Bavaria). The source is shrews, not horses; infected horses do not shed virus.
Horses probably pick up virus from urine, droppings or saliva of infected bicoloured white-toothed shrews, e.g. via contaminated hay, the nose or small wounds. Horses are dead-end hosts: no horse-to-horse spread.
| Method | Sample | Form code |
|---|---|---|
| IFAT* (antibodies) | serum | #2200 |
| PCR* | cerebrospinal fluid (liquor), cooled | #2204 |
Shrews are protected and must not be killed. A definite diagnosis usually needs virus detection in brain tissue after death; blood antibody tests can give false positives.
WNV · Orthoflavivirus nilense (West Nile virus)
Mosquito-borne virus circulating in wild birds, present in Germany since 2018. Most infected horses stay healthy, but some develop severe, sometimes fatal brain and spinal cord disease.
Mosquito-borne RNA virus (flavivirus family)
About 3–15 days after the infected mosquito bite
People are infected by mosquito bites, not by horses. Most have no symptoms; about 20% get flu-like illness and under 1% severe brain inflammation. Protect yourself from mosquitoes too.
Spread by bites of infected mosquitoes (mainly Culex), which pick up the virus from wild birds. Horses and people are dead-end hosts: they do not pass the virus on, so infected horses are no risk to others.
| Method | Sample | Form code |
|---|---|---|
| ELISA IgM (recent infection) | serum | #2916 |
| ELISA IgM and IgG | serum | #2931 |
| PCR | EDTA blood, cerebrospinal fluid (cooled) | #2950 |
About 8% of infected unvaccinated horses develop nervous signs; 30–50% of these die and survivors often keep lasting damage. Cases peak late summer to autumn.
Section 4
Agents found in droppings of foals and adult horses. Send about 50 g of fresh faeces; some tests need cooled overnight transport.
Sampling note on the form: Sample material: faeces, preferably 50 g
Campylobacter spp. (mainly C. jejuni and C. coli)
Gut bacteria carried by many animals and the commonest bacterial cause of food poisoning in people. In horses, their role in causing diarrhoea is still unclear.
Spiral-shaped, Gram-negative gut bacterium
Not established in horses; in people usually 2–5 days
Possible. People get diarrhoea, cramps and fever, rarely nerve or joint complications. Most human cases come from poultry meat; contact with animals is another route. Small children are often affected.
Swallowed with faeces from carrier animals, directly or via contaminated feed, water or surroundings. Birds, poultry and other livestock are main carriers. Healthy horses may carry it unnoticed.
| Method | Sample | Form code |
|---|---|---|
| PCR* | faeces (preferably 50 g) | #5400 |
Survives for a while in cool, damp places but cannot multiply outside an animal. Studies of healthy horses report very different carrier rates.
Clostridioides difficile (formerly Clostridium difficile)
A gut bacterium whose toxins cause sudden, often severe diarrhoea (colitis). Antibiotics, hospital stays and feed changes raise the risk; young foals can also fall ill.
Spore-forming gut bacterium that lives without oxygen; toxins A and B
Unknown; possibly under 24 hours; after antibiotics usually within one week
Possibly. People get C. difficile especially in hospitals and after antibiotics. Horses carry strain types also found in people, but direct spread from horses is not proven. Older and immunocompromised people are most at risk.
Hardy spores are swallowed from faeces or contaminated surroundings. Up to 10% of healthy adult horses and many young foals carry it. Illness follows disturbed gut flora, e.g. antibiotics, stress, transport or surgery.
| Method | Sample | Form code |
|---|---|---|
| PCR for toxin genes A+B° | faeces (preferably 50 g), cooled | #5572 |
Spores survive heat, cold, drying and UV light for long periods. Never spread manure or bedding from sick horses on pasture. Foals can fall ill without antibiotics.
Clostridium perfringens
A normal soil and gut bacterium. Certain toxin-producing strains cause sudden, often bloody diarrhoea that can kill newborn foals within days; adults can develop colitis.
Spore-forming gut bacterium that lives without oxygen; types defined by toxins
Unknown; illness can start within 24 hours; foals usually under 10 days
People commonly get C. perfringens food poisoning from cooked food kept warm too long. No cases caught from horses are documented; basic hygiene around horses with diarrhoea is enough.
Spores are everywhere in soil and faeces. Foals may swallow them from the mare's udder or surroundings. Illness follows overgrowth and toxin production, often after gut upsets such as antibiotics or feed changes.
| Method | Sample | Form code |
|---|---|---|
| PCR for toxin genes° | faeces (preferably 50 g), cooled | #5582 |
Many toxin types occur in healthy horses, so a positive test needs interpretation. Type C is rare in healthy foals. Spores resist heat, cold and drying.
Cryptosporidium spp. (in horses mainly C. parvum and the Cryptosporidium horse genotype)
A tiny parasite that can cause diarrhoea in young foals, often without symptoms. It matters because the main type, C. parvum, also infects people.
Single-celled gut parasite spread by hardy egg-like oocysts
Not established in foals; 2–7 days in calves
Yes; horses are a recognised source. People swallow oocysts from faeces or water and get watery diarrhoea and cramps. Young children and immunocompromised people can become seriously ill.
Foals swallow oocysts from faeces of other foals, mares or calves, or from contaminated water, feed and surroundings. Very few oocysts are needed. Healthy adults can shed them unnoticed.
| Method | Sample | Form code |
|---|---|---|
| ELISA (antigen)° | faeces (preferably 50 g) | #5280 |
Oocysts survive many months in cool, damp places and resist most common disinfectants. In a European study only 0–4.5% of foals were shedding them.
Giardia duodenalis (syn. G. intestinalis, G. lamblia)
A common gut parasite of many animals and people. Horses, especially foals, often carry it without signs; illness in horses is rarely reported.
Single-celled, whip-tailed (flagellate) gut parasite forming hardy cysts
Not established for horses; in animals cysts appear 3–10 days after infection
Possibly. Horses can carry types A and B, which also infect people, but proven spread from animals is rare. People get diarrhoea and cramps, mostly via contaminated water or food.
Cysts passed in faeces are swallowed, directly from infected animals or via contaminated water, feed or surroundings. Foals shed more cysts than older horses.
| Method | Sample | Form code |
|---|---|---|
| ELISA (antigen)° | faeces (preferably 50 g) | #5270 |
Cysts survive weeks to months in cool, damp places but die when dried out. About 10–14% of European foals shed cysts, usually without diarrhoea.
Equine proliferative enteropathy, EPE · Lawsonia intracellularis
Causes equine proliferative enteropathy: a thickened small intestine leaks protein, so weanlings lose weight and develop swellings. Diarrhoea is not always present; mostly autumn–winter.
Bacterium that can only live inside gut lining cells
About 14 days
No known risk to people. The bacterium also infects pigs and some other animals, but no human infections are known.
Not fully known; probably by swallowing faeces of infected animals. Wildlife, rodents and pigs may be sources. The bacterium survives up to two weeks in faeces at mild temperatures.
| Method | Sample | Form code |
|---|---|---|
| PCR | faeces (preferably 50 g) | #5450 |
Mostly weanlings aged 2–8 months. About 5% of exposed foals fall ill. Recovery of body condition and protein can take months; affected yearlings may sell for less.
Equine rotavirus · Rotavirus alphagastroenteritidis (rotavirus A); rotavirus B also found in foals
The most common viral cause of diarrhoea in foals. It spreads extremely fast on studs and causes watery diarrhoea and dangerous dehydration, mainly in foals under two months.
Non-enveloped double-stranded RNA virus (family Sedoreoviridae)
Short: 12–24 hours (AAEP) to 2–6 days (StIKo Vet)
No infections of people caught from horses are documented. Equine-like rotavirus genes have turned up in human strains through gene mixing, so hygiene around sick foals is sensible.
Foals swallow virus from faeces of infected foals, via contaminated feed, hands, boots, tools and stables. Tiny amounts infect; sick foals shed huge numbers, and virus survives months in stables.
| Method | Sample | Form code |
|---|---|---|
| PCR | faeces (preferably 50 g) | #5262 |
The inactivated vaccine (Equip Rotavirus) covers rotavirus A (G3) only and makes disease milder, not impossible; foals keep raised antibodies about 60 days. Rotavirus B is not covered.
Salmonellosis · Salmonella enterica subsp. enterica (many serovars, e.g. Typhimurium)
A gut bacterium causing fever, severe diarrhoea and colic, especially in stressed, sick or hospitalised horses and newborn foals. It spreads easily and can infect people.
Rod-shaped, Gram-negative gut bacterium; more than 2,500 types (serovars)
Hours to days, depending on dose, strain and the horse's resistance
Yes. Salmonella types that make horses ill can seriously sicken people through contact with faeces, hands or equipment. Children, elderly, pregnant and immunocompromised people are most at risk.
Swallowed from faeces of sick or healthy carrier horses, directly or via pasture, feed, water, hands, boots, tools and flies. Stress, transport, colic, surgery or antibiotics make shedding and illness more likely.
| Method | Sample | Form code |
|---|---|---|
| Culture (BE aerobic) + PCR | faeces (preferably 50 g) | #5305 |
Recovered horses may shed for 30–60+ days, a few intermittently for longer. The bacteria have survived 300 days in soil, 9 months in water, 30 months in dried faeces.
Section 5
Culture-based tests on swabs, tissue or blood. They identify which bacteria or fungi grow – and which antibiotics still work.
| Clinical sample – Panel 1: aerobic culture incl. ORSA, resistograms | culture swab, native material | #5043 |
| Clinical sample – Panel 2: aerobic & anaerobic culture incl. ORSA, resistograms | culture swab, native material | #5044 |
| Anaerobic culture | culture swab, native material | #5001 |
| Mycological examination without dermatophytes | culture swab | #5002 |
| Mycological examination incl. dermatophytes | skin scraping | #5046 |
| Blood culture, aerobic, incl. resistograms | BC flask aerobic | #5047 |
| Blood culture, anaerobic | BC flask anaerobic | #5048 |
MRSA / ORSA (methicillin-resistant Staphylococcus aureus) · Staphylococcus aureus, methicillin- (oxacillin-) resistant strains (MRSA = ORSA)
Antibiotic-resistant staph bacteria that horses can carry unnoticed in the nose. They can cause hard-to-treat wound and surgical infections and pass between horses and people.
Gram-positive Staphylococcus aureus strain resistant to many antibiotics
Not defined: carriers may stay healthy; infection follows wounds or surgery
Yes. About 1 in 5 German equine vet staff tested carried horse-type MRSA in the nose; infections are occasional. Risk is higher for people with wounds, diabetes or weak immunity.
Spread mainly by hands and direct contact, between horses and people in both directions, and via equipment. Hospital stays, surgery and earlier antibiotic treatment raise the risk. Most carrier horses stay healthy.
| Method | Sample | Form code |
|---|---|---|
| Aerobic culture incl. ORSA screening + resistograms (Panel 1) | culture swab, native material | #5043 |
| Aerobic & anaerobic culture incl. ORSA + resistograms (Panel 2) | culture swab, native material | #5044 |
Healthy horses carry MRSA rarely (0–6%), but rates are much higher in equine clinics. Most German horse MRSA belong to lineage CC398; spread into the wider community appears rare.
Trichophyton equinum (main), also Microsporum canis, Trichophyton mentagrophytes and other dermatophytes
A common, contagious fungal skin infection causing round bald, scaly patches, often under tack. Rarely dangerous, but spores persist for months and people can catch it.
Thread-like skin fungi that feed on keratin; spread by spores
4–30 days; untreated, usually heals within 1–6 months
Yes. People catch it by touching infected horses or contaminated kit, typically as ring-shaped, scaly skin patches about 1–2 weeks later. Dermatophytes are important zoonotic agents; some species spread to people more readily.
Direct contact with infected horses, or indirectly via spores on grooming kit, tack, rugs, stable fittings, transport vehicles and parasites. Infected hairs are the main source; some horses carry spores without signs.
| Method | Sample | Form code |
|---|---|---|
| Mycological examination incl. dermatophytes (fungal culture) | skin scraping | #5046 |
Spores survive months to years in stables, pasture and shed hair. Recovery speeds healing of later infections but does not prevent them. Fungal culture results take at least three weeks.
Section 6
Worms detected in droppings. Faecal egg counts guide targeted deworming instead of routine treatment.
Strongyles, roundworms, tapeworms, liver fluke · Cyathostominae (small strongyles), Strongylus spp. (large strongyles), Parascaris spp., Anoplocephala perfoliata, Fasciola hepatica
Small redworms occur on practically every horse farm. Heavy worm burdens cause weight loss, diarrhoea or colic, and wormer resistance is widespread, so base worming on dung tests.
Parasitic worms: roundworms (nematodes), tapeworms and flukes
Eggs appear 6 weeks to 7 months after infection, depending on worm type
Liver fluke occasionally infects people who eat raw contaminated water plants, not through horse contact. Equine strongyles, roundworms and tapeworms are not known to infect people.
Worm eggs pass in droppings. Strongyle larvae develop on grass and are swallowed while grazing; roundworm eggs survive years in the environment; tapeworms come via tiny pasture mites, liver fluke via snails on wet pasture.
| Method | Sample | Form code |
|---|---|---|
| Sedimentation and flotation (egg detection) | faeces | #5200 |
The form lists the method, not single species – this sheet groups the worms that method can detect.
Resistance is common: small redworms to benzimidazoles, roundworms to ivermectin-type wormers. One tapeworm treatment yearly in late autumn or winter usually suffices.
Dictyocaulus arnfieldi
A roundworm living in the airways that donkeys carry, usually without signs. Horses grazing with donkeys can develop a stubborn cough that does not respond to antibiotics.
Parasitic roundworm (nematode) living in the airways
About 3 months until larvae appear; time to first cough unclear
Not known to infect people. This lungworm lives in donkeys, horses and mules.
Larvae pass in droppings, mainly from donkeys, and develop on pasture. Horses swallow them while grazing. Adult horses rarely shed larvae, so donkeys are the main source of pasture contamination.
| Method | Sample | Form code |
|---|---|---|
| Migration technique (Baermann) – larvae detection | faeces | #5202 |
In horses the worms rarely mature, so dung tests often miss them; an airway wash by the vet is more reliable.
Section 7
Pathogens carried by ticks. Check your horse after grazing in tall grass and woodland edges.
Equine piroplasmosis · Theileria equi (formerly Babesia equi) and Babesia caballi
Two tick-borne blood parasites that destroy red blood cells, causing fever and anaemia. Recovered horses often stay carriers, and a few horses have now caught it in Germany.
Tick-borne single-celled parasites of red blood cells
T. equi 12–19 days; B. caballi about 10–30 days
Not shown to infect people. Human babesiosis in Europe is mainly caused by other species, such as the cattle parasite Babesia divergens, not horse parasites.
Spread by ticks (Dermacentor, Rhipicephalus, Hyalomma; in Germany especially the spreading ornate dog tick, Dermacentor reticulatus). Also via blood on shared needles, instruments or transfusions, and T. equi from mare to foal.
| Method | Sample | Form code |
|---|---|---|
| PCR (direct detection) | EDTA blood | #1020, 1120 |
| IFAT (antibodies) | serum | #1001, 1101 |
T. equi is more severe and usually persists for life, often even after treatment; B. caballi usually clears within about 4 years. Healthy-looking carriers can infect ticks.
Equine granulocytic anaplasmosis · Anaplasma phagocytophilum (formerly Ehrlichia equi)
A tick-borne bacterium that infects white blood cells and causes sudden high fever in horses. Found wherever the castor bean tick lives, including Germany; antibiotics work well.
Tick-borne bacterium living inside white blood cells
Usually 1–2 weeks after the tick bite; up to 3 weeks reported
Horses do not pass it to people. Humans can catch the same bacterium from tick bites (human granulocytic anaplasmosis), but cases in Germany are very rare.
Spread by bites of Ixodes ticks (in Europe the castor bean tick); wild rodents and deer are reservoirs. Not contagious between horses, except rarely via blood (shared needles) or mare to unborn foal.
| Method | Sample | Form code |
|---|---|---|
| PCR (direct detection) | EDTA blood, tick° | #2302 |
| IFAT (antibodies) | serum | #2300 |
Peaks with tick activity in spring and autumn. Recovered horses are not known to stay carriers and are usually immune for at least two years.
Lyme borreliosis · Borrelia burgdorferi sensu lato (syn. Borreliella); in Europe mainly B. afzelii, B. garinii
A tick-borne bacterium that infects many horses, yet proven illness in horses is rare. Documented problems include eye inflammation, nervous system disease and skin lumps.
Spiral-shaped, tick-borne bacterium (spirochaete)
Not established in horses; antibodies appear about 3–10 weeks after infection
Horses do not infect people, but the same ticks transmit Lyme disease to humans. Check yourself for ticks after yard work; see a doctor for a spreading ring-shaped rash.
Spread by bites of Ixodes ticks (in Europe the castor bean tick); transmission needs hours to days of feeding. Horses are dead-end hosts: no spread between horses or from horses to people.
| Method | Sample | Form code |
|---|---|---|
| PCR (direct detection) | tick°, synovial fluid°, EDTA blood, cerebrospinal fluid, skin biopsy° | #3012 |
Locally up to 30% of ticks in Germany carry Borrelia. Antibodies are common in healthy horses, so a positive blood test alone does not prove disease.
Section 8
Further viruses and parasites the lab tests for – from mild skin or eye infections to notifiable diseases.
Equine herpesvirus 2 · Percavirus equidgamma2 (Equid gammaherpesvirus 2)
A herpesvirus that nearly all horses catch as foals and carry for life. Usually harmless; its role in eye inflammation and airway disease is still debated.
DNA virus (gammaherpesvirus, genus Percavirus)
Not established for horses
No known risk. No human infections are described in the sources consulted.
Through nasal secretions and close contact. Most foals are infected by their mothers or other foals within the first months of life. Carriers shed the virus again from time to time.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab | #1922 |
A positive EHV-2 test does not prove it caused the illness, as healthy horses often carry it. Foals usually test positive by five months of age.
Equine herpesvirus 3 (EHV-3, equine coital exanthema) · Varicellovirus equidalpha3 (Equid alphaherpesvirus 3)
A herpesvirus spread mainly by mating. It causes painful blisters and sores on the genitals of mares and stallions, disrupting the breeding season, but is rarely serious.
DNA virus (alphaherpesvirus, genus Varicellovirus)
About 4–9 days after mating or contact
No known risk. No human infections are described in the sources consulted.
Mainly by mating. Also spread by shared gloves, examination instruments and artificial vaginas during breeding work and artificial insemination. Horses carrying the dormant virus can shed it again later.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab | #1932 |
Skin sores usually heal within 2–3 weeks. Fertility and pregnancies are not affected. The virus can lie dormant in the body and flare up again later.
Equine herpesvirus 5 · Percavirus equidgamma5 (Equid gammaherpesvirus 5)
A herpesvirus most horses carry without problems. In some horses, mainly middle-aged ones, it is linked to a serious lung-scarring disease (EMPF) causing cough and weight loss.
DNA virus (gammaherpesvirus, genus Percavirus)
Not established for horses
No known risk. No human infections are described in the sources consulted.
Probably through nasal secretions and close contact, as with EHV-2. Foals become infected early in life, before weaning. Infected horses carry the virus for life.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab | #1952 |
About half of horses with EMPF survive. A positive EHV-5 test alone means little, because many healthy horses carry the virus.
Equine adenovirus 1 (EAdV-1) · Mastadenovirus equi (equine adenovirus 1; formerly Equine mastadenovirus A)
A widespread respiratory virus, often found in healthy horses and usually mild. It can be deadly for foals born with an inherited immune defect (SCID), seen in Arabians.
DNA virus (adenovirus, genus Mastadenovirus)
Not established for horses
No known risk. No human infections are described in the sources consulted.
By droplets and close contact with nose and eye secretions; the virus is also shed in droppings. Healthy-looking horses can shed it, sometimes for weeks after infection.
| Method | Sample | Form code |
|---|---|---|
| PCR | dry swab | #1562 |
Healthy horses usually recover within 7–10 days. SCID foals often die of adenovirus pneumonia once maternal antibodies wane, at about 2–5 months.
Equine infectious anaemia (EIA, swamp fever) · Lentivirus equinfane (equine infectious anaemia virus, EIAV)
A blood-borne virus disease with no cure or vaccine. Infected horses carry the virus for life, often without signs, so they are a lasting source of infection.
Retrovirus of the lentivirus group; infection lasts for life
Usually 1–3 weeks, but can be up to 3 months
People cannot catch EIA: WOAH and FLI state the virus poses no risk to human health.
Spread through blood: mainly by horseflies and stable flies carrying blood between horses within about 200 m, and by shared needles, instruments or blood products. Also from mare to foal before birth.
| Method | Sample | Form code |
|---|---|---|
| Coggins test (AGID, antibodies) | serum | #1200 |
| ELISA (antibodies) | serum | #1202 |
Cases in Germany are sporadic and often linked to imported horses. Donkeys, mules and zebras can also be infected.
Equine protozoal myeloencephalitis · Sarcocystis neurona (rarely Neospora hughesi)
A parasite of the brain and spinal cord found in the Americas, spread by opossums. Elsewhere, including Europe, it affects only horses that have lived there.
Single-celled parasite attacking the brain and spinal cord
Not established for natural infection in horses
Not known to infect people. Human Sarcocystis infections come from other species, e.g. in raw beef or pork, not from horses.
Horses swallow parasite sporocysts (egg-like stages) in feed or water soiled by opossum droppings. Horses are dead-end hosts: EPM does not spread between horses.
| Method | Sample | Form code |
|---|---|---|
| Western blot* (antibodies) | serum | #2800 |
Many American horses have antibodies, but few fall ill. Stress and young age raise the risk. With treatment about 60% improve; under a quarter recover fully.